Monday, November 12, 2012

Types of Diabetes and How to Control It

It will withal see what to be aware of after athletic performance (e.g. delay onset hypoglycaemia). In the normal athlete, resting glucose levels are kept deep down a narrow range by the complex interaction of the autonomic nervous system and hormones, including insulin, glucagon and adrenalin (Dawson, 2005; Draznin, 2000). With usance, glucose production by the liver is upregulated and muscles take up glucose from the blood stream and also use endogenous glycogen as a terminate source. Non-esterified fatty acids can be mobilized to provide extra substratum for working muscles. Insulin production is downregulated during commit to prevent hypoglycemia because of change magnitude muscle uptake of glucose from the bloodstream. When action is over, the body is relatively depressed of glucose and insulin counter-regulatory mechanisms stimulate gluconeogenesis by the liver. This leads to an increase in blood glucose, and and so an increase in insulin production.

In Type 1 diabetics, there is no endogenous insulin production and so during exercise hypoglycemia or hyperglycemia

whitethorn occur (Dawson, 2005). If there is insulin excess, hypoglycemia whitethorn occur either during or after exercise because hepatic gluconeogenesis is inhibited and increased glucose uptake through the synergy of Glut-4 receptor stimul


hammering of the toes was only instal in two of 8 neuropathic subjects. Although most often it is the sensorial neuropathy that is emphasized in diabetics, this study showed that the motor neuropathy in the feet is profound, and may play a major role in postural stability. This is crucially important to the diabetic athlete, and needs to be assessed forward serious injury occurs due to this instability, e.g. a fall, broken bones, or serious sprains.

American Diabetes Association. (2005). Foot complications. Retrieved Dec. 10, 2005 from

1) avoid exercise if temperance glucose levels are above 250 mg/dL and ketosis is present, or above 300 mg/dL whether or not ketosis is present

decrease in muscle tissue distally compared to controls. Muscle composed only 8.3 + 2.
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9 percent of the total cull cross-sectional area in diabetics with neuropathy compared to 30.8 + 3.9 percent in controls. No significant differences were found between the groups in metatarso-phalangeal or proximal and distal interphalangeal joint angles of the second ray. Clawing/

http://www.activehealth.co.nz/diabetes.htm

Boyajian-O'Neill, L., Cardone, D., Dexter, W., DiFiori, J., Fields, B., Jones, D. Et al. (2004). Determining clearance during the preparticipation evaluation. The Physician and Sports Medicine, 32(11). Retrieved Dec. 10, 2005 from

Athletes with type 1 diabetes need to tailor their insulin, unsound and food intake to meet their individual response, the duration of their exercise, the glitz of the exercise and the exercise environment (Dawson, 2005). They need to regularly admonisher their blood glucose levels, particularly when beginning exercise. It is important for the Type 1 diabetic to control nutritional content before, during and after exercise to avoid adverse reactions. A 50 percent low-down glycemic index carbohydrate meal two hours before exercise is recommended, and carbohydrate intake should be maintained during exercise. After exercise and before slee
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